User profiles for Pietro Baldelli
Pietro BaldelliUniversity of Genova, Dept. Exp. Med. Verified email at unige.it Cited by 5503 |
[HTML][HTML] Acute stress increases depolarization-evoked glutamate release in the rat prefrontal/frontal cortex: the dampening action of antidepressants
…, VS Barbiero, T Bonifacino, A Mallei, P Baldelli… - PloS one, 2010 - journals.plos.org
Background Behavioral stress is recognized as a main risk factor for neuropsychiatric
diseases. Converging evidence suggested that acute stress is associated with increase of …
diseases. Converging evidence suggested that acute stress is associated with increase of …
SYN1 loss-of-function mutations in autism and partial epilepsy cause impaired synaptic function
…, M Fadda, A Corradi, P Baldelli… - Human molecular …, 2011 - academic.oup.com
Several genes predisposing to autism spectrum disorders (ASDs) with or without epilepsy
have been identified, many of which are implicated in synaptic function. Here we report a …
have been identified, many of which are implicated in synaptic function. Here we report a …
Lack of synapsin I reduces the readily releasable pool of synaptic vesicles at central inhibitory synapses
P Baldelli, A Fassio, F Valtorta… - Journal of …, 2007 - Soc Neuroscience
Synapsins (Syns) are synaptic vesicle (SV) phosphoproteins that play a role in neurotransmitter
release and synaptic plasticity by acting at multiple steps of exocytosis. Mutation of SYN …
release and synaptic plasticity by acting at multiple steps of exocytosis. Mutation of SYN …
Microbiota-gut brain axis involvement in neuropsychiatric disorders
Introduction: The microbiota-gut brain (MGB) axis is the bidirectional communication
between the intestinal microbiota and the brain. An increasing body of preclinical and clinical …
between the intestinal microbiota and the brain. An increasing body of preclinical and clinical …
[PDF][PDF] PRRT2 is a key component of the Ca2+-dependent neurotransmitter release machinery
Heterozygous mutations in proline-rich transmembrane protein 2 (PRRT2) underlie a group
of paroxysmal disorders, including epilepsy, kinesigenic dyskinesia, and migraine. Most of …
of paroxysmal disorders, including epilepsy, kinesigenic dyskinesia, and migraine. Most of …
Protein kinase A-mediated synapsin I phosphorylation is a central modulator of Ca2+-dependent synaptic activity
…, G Ferrari, HT Kao, F Benfenati, P Baldelli… - Journal of …, 2006 - Soc Neuroscience
Protein kinase A (PKA) modulates several steps of synaptic transmission. However, the
identification of the mediators of these effects is as yet incomplete. Synapsins are synaptic …
identification of the mediators of these effects is as yet incomplete. Synapsins are synaptic …
[HTML][HTML] Emergent functional properties of neuronal networks with controlled topology
The interplay between anatomical connectivity and dynamics in neural networks plays a key
role in the functional properties of the brain and in the associated connectivity changes …
role in the functional properties of the brain and in the associated connectivity changes …
PRRT2 controls neuronal excitability by negatively modulating Na+ channel 1.2/1.6 activity
See Lerche (doi: 10.1093/brain/awy073 ) for a scientific commentary on this article. Proline-rich
transmembrane protein 2 (PRRT2) is the causative gene for a heterogeneous group of …
transmembrane protein 2 (PRRT2) is the causative gene for a heterogeneous group of …
Opposite changes in glutamatergic and GABAergic transmission underlie the diffuse hyperexcitability of synapsin I–deficient cortical networks
Synapsins (Syns) are synaptic vesicle (SV) phosphoproteins that play a role in synaptic
transmission and plasticity. Mutation of the SYN1 gene results in an epileptic phenotype in …
transmission and plasticity. Mutation of the SYN1 gene results in an epileptic phenotype in …
TBC1D24 regulates neuronal migration and maturation through modulation of the ARF6-dependent pathway
…, E Pallesi-Pocachard, P Baldelli… - Proceedings of the …, 2014 - National Acad Sciences
Alterations in the formation of brain networks are associated with several neurodevelopmental
disorders. Mutations in TBC1 domain family member 24 (TBC1D24) are responsible for …
disorders. Mutations in TBC1 domain family member 24 (TBC1D24) are responsible for …