Nicotine exposure in utero negatively affects neuronal growth, differentiation, and synaptogenesis.
We used rhythmic brainstems slices and immunohistochemistry to determine how …

Regulation of GABAergic signaling through nicotinic acetylcholine receptor (nAChR) activation
is critical for neuronal development. Here, we test the hypothesis that chronic episodic …

… Lila Buls Wollman is a first-year postdoctoral fellow at the Center for … Wollman's research
interests include respiratory neural and … Wollman is investigating strategies to improve breathing …

Phrenic long-term facilitation (LTF) is a sustained increase in phrenic motor output occurring
after exposure to multiple (but not single) hypoxic episodes. Ampakines are a class of drugs …

We tested the hypothesis that nicotine exposure in utero and after birth [developmental
nicotine exposure (DNE)] disrupts development of glycinergic synaptic transmission to …

Developmental nicotine exposure (DNE), alters brainstem neurons that control breathing,
including hypoglossal motor neurons (XIIMNs), which innervate the tongue. Here, we tested …

Nicotine exposure during the fetal and neonatal periods [developmental nicotine exposure (DNE)]
is associated with ineffective upper airway protective reflexes in infants. This could be …

Nicotinic acetylcholine receptors (nAChRs) are expressed throughout the central respiratory
network, including postsynaptically on pattern generating neurons and motor neurons, and …

Cotinine, the primary metabolite of nicotine, can be detected in the blood of infants born to
mothers who used nicotine products during pregnancy and breast feeding, and is used as a …

Developmental nicotine exposure (DNE) is known to cause abnormal development of multiple
brain regions and results in impaired control of breathing and altered behaviors that rely …