ArticleBrain ethanol in AA, ANA, and Wistar rats monitored with one-minute microdialysis
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2014, Brain ResearchCitation Excerpt :The samples were stored at −20 °C until analysis. They were analyzed with headspace gas chromatography (Perking Elmer, GC 8410 gas chromatography and HS 40 headspace autosampler, Shelton, CT, USA) as described elsewhere (Nurmi et al., 1994). Group means and standard errors were calculated for each animal group comprising 6–10 animals.
Brain-derived neurotrophic factor expression after acute administration of ethanol
2012, European Journal of PharmacologyCitation Excerpt :The samples were stored at − 20 °C until analysis. They were analyzed with headspace gas chromatography (Perking Elmer, GC 8410 gas chromatography with HS 40 headspace autosampler, Shelton, Connecticut, USA), a standard method in this laboratory (cf. Eriksson, 1973; Nurmi et al., 1994). Group means and standard errors were calculated for each animal group comprising 7–10 animals.
Intra-amygdala inhibition of ERK <inf>1/2</inf> potentiates the discriminative stimulus effects of alcohol
2012, Behavioural Brain ResearchCitation Excerpt :Briefly, immediately following intragastric gavage (IG) administration alcohol (1 g/kg) or water rats were placed in the chambers and after a 10 min delay the house light was illuminated and both levers were extended into the chamber (beginning of the 15-min session). IG administration of alcohol results in rapid brain alcohol concentrations [33], and the 10 min time point corresponds to the ascending limb of peak blood and brain alcohol concentrations [33,34]. Following alcohol administration, completion of an FR10 on the alcohol-appropriate lever resulted in the availability of the sucrose (10%, w/v) solution.
The shell of the nucleus accumbens has a higher dopamine response compared with the core after non-contingent intravenous ethanol administration
2008, NeuroscienceCitation Excerpt :Unfortunately, the dose-dependence of this effect has not been firmly established with one group showing that 0.5 g/kg (i.p.) ethanol did not produce place conditioning (Biala and Kotlinska, 1999) whereas others have reported success using this dose and route of administration (Bozarth, 1990; Zhu et al., 2007). The peak brain concentrations after 0.5 g/kg ethanol (i.p.) should be approximately 17 mM (Nurmi et al., 1994), and our present data suggest that this concentration will produce a modest, threshold effect on dopamine release. Taken together, it is tempting to speculate that ethanol-induced conditioned place preference in rats is largely independent of an accumbal dopamine response.
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2008, Physiology and BehaviorChapter 3.3 Improvement of the temporal resolution of brain microdialysis: sampling in seconds
2006, Handbook of Behavioral NeuroscienceCitation Excerpt :Kehr has used o-phthalaldehyde derivatization with fluorescence detection combined with microbore columns to measure glutamate and aspartate in brain dialysates every minute (Kehr, 1998). In any event, microbore columns have been used to measure drugs such as ethanol (Nurmi et al., 1994) and acetaminophen (Chen and Lunte, 1995) in plasma dialysates every minute. Newton and Justice (1994) measured extracellular levels of dopamine in brain dialysates every minute, using microbore columns.