Regulation of feline aggression by the bed nucleus of stria terminalis
References (20)
- et al.
Effects of electrical stimulation of the amygdala upon trigeminal sensory fields established during hypothalamically elicited quiet biting attack in the cat
Brain Res
(1980) - et al.
An analysis of the mechanisms underlying septal area control of hypothalamically elicited aggression in the cat
Brain Res
(1984) - et al.
The organization of the hypothalamic pathways mediating affective defense behavior in the cat
Brain Res
(1985) - et al.
Differential control of aggression by the midbrain
Exp Neurol
(1984) - et al.
Forebrain structures regulating flight behavior in the cat
Brain Res Bull
(1985) - et al.
Effects of electrical stimulation of the pyriform cortex upon hypothalamic attack behavior in cats
Brain Res
(1972) - et al.
Septal and amygdalar efferents to the hypothalamus which facilitate hypothalamically elicited intraspecific aggression and associated hissing in the cat. An autoradiographic study
Brain Res
(1980) - et al.
A 14C-2-deoxyglucose analysis of the functional neural pathways of the limbic forebrain in the rat. I. The amygdala
Brain Res Rev
(1983) Predatory behavior in the cat elicited by lower brainstem and hypothalamic stimulation: a comparison
Brain Behav Evol
(1977)- et al.
Effects of experimental temporal lobe seizures upon hypothalamically elicited aggressive behavior in the cat
Brain Res
(1985)
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2018, Cell MetabolismAggression and anxiety in adolescent AAS-treated hamsters: A role for 5HT<inf>3</inf> receptors
2015, Pharmacology Biochemistry and BehaviorCitation Excerpt :Anabolic/androgenic steroid exposure specifically decreased the presence of 5HT3 receptors in the BNST, MeA, CeA, and AH of aggressive hamsters. The BNST, MeA, and CeA play a role in the sensory interpretation and olfactory communication integration in the aggressive behavioral sequelae (Amaral et al., 2003; Bamshad et al., 1997; Shaikh et al., 1986; Vinkers et al., 2010), while also playing a role in the expression of anxiety-related behaviors (Davis, 1998; Kalin et al., 2004; Liebsch et al., 1995; Pandey et al., 2003; Pratt, 1992), and are considered a critical part of the extended amygdala, regulating emotional behaviors (Cassell et al., 1999; McDonald, 2003). We also showed that the AH had significantly fewer 5HT3 receptors and, accordingly, is known to share reciprocal connections with the BNST, MeA and CeA as well as other regions of the brain implicated in both aggression and anxiety (Carrillo et al., 2009, 2011; Delville et al., 2000).
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2015, Behavioural Brain ResearchThe neural background of hyper-emotional aggression induced by post-weaning social isolation
2012, Behavioural Brain ResearchCitation Excerpt :Increased activations were observed in other amygdala regions except for the central amygdala. These extended amygdala regions, e.g. the bed nucleus of stria terminalis, and the lateral-basolateral amygdala complex also promote aggressive behavior [40–43] that perhaps contributes to the heightened aggression observed in socially isolated rats. Other regions playing indirect or modulator roles in aggression control also showed interesting changes.