Clinical profile of the study participants
Subjectno. | Age | Gender | Handedness | SOZ | MRI | Brain pathology | Language laterality(method) | Stimulation mapping overlap | vIQ | Verbalmemorydeficits |
---|---|---|---|---|---|---|---|---|---|---|
1001 | 48 | F | R | Right TC | Normal | Gliosis | L (fMRI) | - | 81 | None |
1006 | 20 | F | R | Right FC | MCD | Gliosis | L (fMRI) | - | 91 | None |
1016 | 31 | F | R | Left FC | Normal | Gliosis | - | None | 71 | None |
1018 | 47 | M | L | Left FC,left FPC | Normal | - | L (fMRI) | - | 85 | None |
1020 | 48 | F | L | Right TC,right FC | Abnormal | Gliosis | L (fMRI) | - | 98 | Mild |
1022 | 24 | M | R | AtrophyGliosis/encephalomalacia | - | L (fMRI) | - | 81 | None | |
1024 | 36 | F | R | Right OPC | Normal | Gliosis | L (unknown) | - | 100 | None |
1026 | 24 | F | R | Left aTCleft OC | MTS, gliosis | - | Bilateral (Wada) | - | 112 | None |
1027 | 48 | M | R | Right TC right ICright/left FC | Abnormal | - | L (fMRI) | - | 93 | None |
1028 | 27 | F | R | Right MTL | Abnormal | CD, Gliosis | L (Wada) | - | 103 | None |
1029 | 33 | F | R | Left FC | Abnormal | - | - | - | 108 | Mild |
1030 | 23 | M | L | Left MTL | Normal | Gliosis | L (fMRI) | - | 106 | None |
1031 | 24 | M | R | Right FC right TC | Abnormal | - | L (aphasia) | - | 110 | Moderate |
1033 | 31 | F | R | Right TC | Atrophy | - | L (Wada) | - | 85 | None |
1036 | 49 | M | L | Left aTC,left MTL | MTS | HS | Bilateral (Wada) | - | 93 | Moderate |
1042 | 27 | F | L | Right TC | MCD | - | R (fMRI) | None | 114 | None |
1050 | 20 | M | R | Left PC | Neoplasm | DNET | Bilateral (Wada) | None | 95 | Mild |
1060 | 36 | F | R | Right TC | Normal | Gliosis | L (Wada) | - | 95 | Mild |
1069 | 26 | M | R | Left FC | MCD | - | L (Wada) | - | - | Mild |
1111 | 20 | M | R | Left TC left OPCleft OC | Gliosis | Gliosis | L (fMRI) | - | 108 | None |
1176 | 41 | F | R | Right MTL right IC | MTS | - | L (Wada) | - | 85 | Moderate |
1177 | 23 | F | R | Left TC | TS | - | L (aphasia) | None | 87 | Moderate |
Patient demographic data are presented together with clinical observations from structural MRI, clinically identified seizure onset zones (SOZs), pathology for those subjects who underwent respective surgery, hemispheric laterality of language functions together with the method of determination (“aphasia” means that the determination was done based on an identified lesion/pathology in a specific hemisphere), overlap of the stimulating electrodes with the language areas for patients who have undergone cortical stimulation mapping (“-”; means that the stimulation mapping was not performed or the report was not available), verbal IQ (vIQ), and the clinical qualitative description of verbal memory deficits as concluded in the neuropsychological assessment. FC, frontal cortex; PC, parietal cortex; OC, occipital cortex; IC, insular cortex; aTC, anterior TC; MTL, mesial temporal lobe; TPC, temporo-parietal cortex; FPC, fronto-parietal cortex; OPC, occipito-parietal cortex; CD, cortical dysplasia; HS, hippocampal sclerosis; MCD, malformation of cortical development; MTS, mesial temporal sclerosis; PMG, polymicrogyria; DNET, dysembryoplastic neuroepithelial tumor.