Article Information
PubMed
Published By
History
- Received February 13, 2021
- Revision received May 10, 2021
- Accepted May 14, 2021
- Published online May 25, 2021.
Copyright & Usage
Copyright © 2021 Runyan et al. This is an open-access article distributed under the terms of the Creative Commons Attribution 4.0 International license, which permits unrestricted use, distribution and reproduction in any medium provided that the original work is properly attributed.
Author Information
- 1Program in Neuroscience, Graduate School of Biomedical Science and Professional Studies, Drexel University College of Medicine, Philadelphia, PA 19129
- 2Department of Neurobiology and Anatomy, Drexel University College of Medicine, Philadelphia, PA 19129
- 3Department of Anesthesiology and Critical Care Medicine, Children’s Hospital of Philadelphia, Philadelphia, PA 19104
- Correspondence should be addressed to Ramesh Raghupathi at rr79{at}drexel.edu.
Author contributions
Author contributions: A.R., D.L., J.W.H., and R.R. designed research; A.R. and D.L. performed research; J.R.B. contributed unpublished reagents/analytic tools; A.R., D.L., J.W.H., J.R.B., and R.R. analyzed data; A.R., D.L., J.W.H., J.R.B., and R.R. wrote the paper.
↵* A.R. and D.L. contributed equally to this work.
Disclosures
The authors declare no competing financial interests.
This work weas supported in part by the National Institutes of Health Grant R01 NS110898 (to J.W.H. and R.R.) and Commonwealth Universal Research Enhancement from the Pennsylvania Department of Health Grants SAP 410-007-9710 (to R.R.) and SAP 410-007-7079 (R.R.).
Funding
HHS | NIH | Eunice Kennedy Shriver National Institute of Child Health and Human Development (NICHD)
HD061963HHS | NIH | National Institute of Neurological Disorders and Stroke (NINDS)
NS110989PA Department of Health
SAP4100079710; SAP4100077079
Other Version
- You are viewing the most recent version of this article.
- previous version (May 25, 2021).
Online Impact
Article usage
In this issue
Intranasal Administration of Oxytocin Attenuates Social Recognition Deficits and Increases Prefrontal Cortex Inhibitory Postsynaptic Currents following Traumatic Brain Injury
