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Research ArticleResearch Article: New Research, Disorders of the Nervous System

KCC2 Chloride Transport Contributes to the Termination of Ictal Epileptiform Activity

Volodymyr I. Dzhala and Kevin J. Staley
eNeuro 25 November 2020, 8 (2) ENEURO.0208-20.2020; DOI: https://doi.org/10.1523/ENEURO.0208-20.2020
Volodymyr I. Dzhala
1Department of Neurology, Massachusetts General Hospital, Boston, MA 02114
2Harvard Medical School, Boston, MA 02114
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Kevin J. Staley
1Department of Neurology, Massachusetts General Hospital, Boston, MA 02114
2Harvard Medical School, Boston, MA 02114
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Article Information

DOI 
https://doi.org/10.1523/ENEURO.0208-20.2020
PubMed 
33239270
Published By 
Society for Neuroscience
History 
  • Received May 19, 2020
  • Revision received November 2, 2020
  • Accepted November 3, 2020
  • Published online November 25, 2020.
Copyright & Usage 
Copyright © 2020 Dzhala and Staley This is an open-access article distributed under the terms of the Creative Commons Attribution 4.0 International license, which permits unrestricted use, distribution and reproduction in any medium provided that the original work is properly attributed.

Author Information

  1. Volodymyr I. Dzhala1,2 and
  2. Kevin J. Staley1,2
  1. 1Department of Neurology, Massachusetts General Hospital, Boston, MA 02114
  2. 2Harvard Medical School, Boston, MA 02114
  1. Correspondence should be addressed to Volodymyr I. Dzhala at vdzhala{at}mhg.harvard.edu or Kevin J. Staley at staley.kevin{at}mgh.harvard.edu.
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Author contributions

  1. Author contributions: V.I.D. and K.J.S. designed research; V.I.D. performed research and analyzed data; V.I.D. and K.J.S. wrote the paper.

Disclosures

  • The authors declare no competing financial interests.

  • This study was supported by the United States of America National Institutes of Health and National Institute of Neurological Disorders and Stroke grant R01 NS040109 (K.J.S.).

Funding

  • HHS | NIH | National Institute of Neurological Disorders and Stroke (NINDS)

    R01 NS040109

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  • You are viewing the most recent version of this article.
  • previous version (November 25, 2020).

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AbstractFullPdf
Nov 2020159078
Dec 20203420129
Total 20205010207
Jan 202198039
Feb 2021115030
Mar 20217530888
Apr 20213922939
May 20211722522
Jun 20211018722
Jul 2021718617
Aug 2021419219
Sep 2021612417
Oct 2021511340
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Dec 202119813
Total 20213791756370
Jan 2022512334
Feb 202266718
Mar 202227927
Apr 202206315
May 202209323
Jun 202229724
Jul 202226615
Aug 202218024
Sep 202226427
Oct 202258220
Nov 202226414
Dec 202216510
Total 202228943251
Jan 202307929
Feb 202307310
Mar 20231868
Apr 202318923
May 202308315
Jun 202309815
Jul 2023310322
Aug 202309331
Sep 202336014
Total 20238764167
Total9163463995
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eneuro: 8 (2)
eNeuro
Vol. 8, Issue 2
March/April 2021
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KCC2 Chloride Transport Contributes to the Termination of Ictal Epileptiform Activity
Volodymyr I. Dzhala, Kevin J. Staley
eNeuro 25 November 2020, 8 (2) ENEURO.0208-20.2020; DOI: 10.1523/ENEURO.0208-20.2020

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KCC2 Chloride Transport Contributes to the Termination of Ictal Epileptiform Activity
Volodymyr I. Dzhala, Kevin J. Staley
eNeuro 25 November 2020, 8 (2) ENEURO.0208-20.2020; DOI: 10.1523/ENEURO.0208-20.2020
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Keywords

  • chloride
  • CLP257
  • GABA
  • KCC2
  • seizure
  • VU0463271

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