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PubMed
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History
- Received June 23, 2015
- Revision received August 25, 2015
- Accepted August 29, 2015
- Published online September 8, 2015.
Copyright & Usage
Copyright © 2015 Rybak et al. This is an open-access article distributed under the terms of the Creative Commons Attribution 4.0 International, which permits unrestricted use, distribution and reproduction in any medium provided that the original work is properly attributed.
Author Information
- Ilya A. Rybak,
- Kimberly J. Dougherty and
- Natalia A. Shevtsova
- Department of Neurobiology and Anatomy, Drexel University College of Medicine, Philadelphia, Pennsylvania 19129
- Correspondence should be addressed to Dr. Ilya A. Rybak, Department of Neurobiology and Anatomy, Drexel University College of Medicine, 2900 Queen Lane, Philadelphia, PA 19129. E-mail: rybak{at}drexel.edu.
Author contributions
Author Contributions: I.A.R., K.J.D., and N.A.S. designed research; I.A.R., K.J.D., and N.A.S. performed research; I.A.R., K.J.D., and N.A.S. wrote the paper.
Disclosures
The authors declare no competing financial interests.
This study was supported by National Institutes of Health Grants NS081713 and NS090919 to I.A.R.; and NS095366 to K.J.D.
Funding
NIH/NINDS
R01 NS081713NIH/NINDS
R01 NS090919NIH/NINDS
R01 NS095366
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In this issue
Organization of the Mammalian Locomotor CPG: Review of Computational Model and Circuit Architectures Based on Genetically Identified Spinal Interneurons
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- Article
- Visual Overview
- Abstract
- Significance Statement
- Introduction
- Models of the locomotor CPG
- Spinal interneurons identified from developmental and genetic studies
- Spinal interneurons involved in locomotor rhythm generation: Shox2 cells
- Ipsilaterally projecting, excitatory V2a interneurons
- Flexor–extensor asymmetry in the organization of locomotor activity
- Left–right coordination of activity in the spinal cord: frequency-dependent role of V0D and V0V CINs
- Axon guidance and left–right coordination: role of Netrin-1, DCC, and EphA4
- Flexor–extensor alternation: role of V1 and V2b interneurons
- Conclusions
- Footnotes
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